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Background More than one-third of U.S. adults are obese, incurring an increased risk of heart failure. Increased body mass index is associated with left-ventricular (LV) diastolic dysfunction, which may be one of the pathophysiological links between obesity and heart failure. Mouse models can elucidate molecular mechanisms that underlie cardiovascular disease. In this study we used high temporal-resolution

High temporal-resolution phase-contrast MRI demonstrates impaired left-ventricular diastolic relaxation in mice fed a high-fat high-sucrose diet