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Background Pulmonary hypertension (PH) is a progressive disease that results in right ventricular dysfunction through increased resistive afterload and pulmonary arterial (PA) stiffening. The ventricles are connected at the interventricular sulcus, share an interlaced network of muscle fibers, and are hemodynamically dependent upon each other via the interventricular septum (IVS). Thus, the left ventricle (LV) plays a major role in RV contractile performance and may also be affected in this disease via ventricular-ventricular interaction. The relationship between hemodynamic parameters and torsion rate is unknown. We hypothesized that LV torsion rate is reduced in pediatric PH and is concomitant with hemodynamic markers of PH, and that this is due to the impact of prolonged increased impedance, resulting in reduced RV performance.

Left Ventricular torsion rates by CMR correlate with invasively-derived hemodynamic data in pediatric pulmonary hypertension