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Background Excess of cardiovascular (CV) morbidity and mortality in RA patients is not fully explained by traditional CV risk factors. Evidence suggests the presence of inflammation-induced vascular and myocardial injury and dysfunction, leading to premature atherosclerosis and heart failure. Aortic stiffness, an independent predictor of cardiac events and a marker of pulsatile LV afterload, improves with anti-tumor necrosis factor-alpha (antiTNF) therapy in these patients; however, the effects on determinants of systolic function remain undetermined.

journal of cardiovascular magnetic resonance

Unravelling the phenotype of cardiovascular inflammation with magnetic resonance imaging: detecting the change with anti-TNF treatment in patients with rheumatoid arthritis