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tertile) compared to low numbers (lower tertile) of CM (mean relative upslope in upper tertile 7.5 versus mid tertile 5.8 versus lower tertile 5.2, p=0.014). Also, patients with high numbers of CM had a larger infarct size (9.17 vs. 6.51 vs. 3.89 g/m 2 , p<0.001), more segments with microvascular obstruction at LGE (15.9 vs. 8.2 vs. 2.2% of segments, p=0.001), and greater impairment of ejection fraction (37.0 vs. 42.0 vs. 46.5%, p=0.001) than patients with low numbers of CM. No such relations were found for NCM. Figure 1.

Impaired myocardial perfusion on CMR is associated with increased numbers of classical monocytes in STEMI patients treated by primary PCI