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Rheumatoid arthritis (RA) and psoriatic arthritis (PsA) are immune-mediated diseases which partly share clinical features, but differentiate in the mechanisms which lead to aberrant immune responses. Natural killer (NK) cells tune the innate immune response depending on the integration of signals coming from a complex network of activating and inhibitory surface receptors.

journal of translational medicine

Extensive natural killer cell receptor phenotyping on NK and T cells discloses differences in RA and PsA, potentially mirroring diverse immunoregulatory functions