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Background  The transcription factor At MYBR1  ( MYB44 ) is a member of the MYB family of transcription factors and is expressed throughout the plant life cycle and especially in senescing and wounded leaves. It has previously been shown to be involved in responses to abiotic stress and is regulated by phosphorylation.    Results  When  MYBR1  was over-expressed under the control of the constitutive 35S promoter in  Arabidopsis thaliana  (Ox MYBR1 ), leaf senescence was delayed. In contrast loss-of-function  mybr1  plants showed more rapid chlorophyll loss and senescence. The  MYBR1  promoter strongly drove  β-GLUCURONIDASE  reporter gene expression in tissues immediately after wounding and many wounding/pathogenesis genes were downregulated in Ox MYBR1 . Ox MYBR1  plants were more susceptible to injury under water stress than wildtype, which was correlated with suppression of many ABA inducible stress genes in Ox MYBR1 . Conversely,  mybr1  plants were more tolerant of water stress and exhibited reduced rates of water loss from leaves. MYBR1 physically interacted with ABA receptor PYR1-LIKE8 (PYL8) suggesting a direct involvement of MYBR1 in early ABA signaling.  MYBR1  appears to exhibit partially redundant functions with At MYBR2  ( MYB77 ) and double  mybr1  X  mybr2  mutants exhibited stronger senescence and stress related phenotypes than single  mybr1  and  mybr2  mutants.    Conclusions   MYBR1  is a negative regulator of ABA, stress, wounding responses and blocks senescence. It appears to have a homeostatic function to maintain growth processes in the event of physical damage or stress.

Multiple roles of the transcription factor AtMYBR1/AtMYB44 in ABA signaling, stress responses, and leaf senescence