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Background Activation of the nitric oxide – soluble guanylate cyclase – cyclic guanosine monophosphate (NO-sGC-cGMP) pathway can induce potent cardioprotection-like effects against ischemia-reperfusion injury. We investigated the effects of pharmacological preconditioning with cinaciguat (BAY 58-2667), a novel sGC-activator on myocardial and coronary vascular function during reperfusion in a canine model of cardioplegic arrest and extracorporal circulation.

bmc pharmacology

Pharmacological preconditioning with the guanylate cyclase activator cinaciguat (BAY 58-2667) protects against reperfusion injury after cardiopulmonary bypass