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Trigeminal autonomic cephalalgias (TACs) are highly disabling primary headaches that involve activation of trigeminovascular neurons and their reflex connection with the parasympathetic outflow to the cranial vasculature, via the superior salivatory nucleus (SuS) in the brainstem. Our understanding of TAC pathophysiology, and how and where treatments act, is hindered by a lack of animal models.

A translational in vivo model of trigeminal autonomic cephalalgias – therapeutic characterization with brainstem stimulation