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Background Migraine is characterized pathophysiologically by an interictal habituation deficit of information processing, responsible for cortical hyperresponsiveness, which normalizes during attacks [1]. Furthermore, descending pain control systems (DPCSs) can be impaired in episodic and chronic migraine [3,4]. Noninvasive neuromodulation of the cerebral cortex may be effective in migraine prophylaxis [2] including, in chronic migraine, modulation of the dorsolateral prefrontal cortex (DLPFC) that is part of the DPCSs [5].

A multidisciplinary approach to the functional abnormalities of the migrainous brain and non-invasive interventions to treat them