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Hemochromatosis is a common inherited disease of iron overload that results from mutations in the HFE gene (1). It has long been known that basic processes of body iron homeostasis, and in particular intestinal iron absorption, are regulated aberrantly in hemochromatosis, so an understanding of how HFE functions in cellular and body iron homeostasis may hold the key to dissecting these essential physiological processes. However, despite a growing body of elegant experimentation, the recalcitrant HFE molecule has yet to reveal all its secrets.

Of metals, mice, and men: what animal models can teach us about body iron loading