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ing limb of Henle's loop, resulting in the delivery of a larger volume of fluid of lower sodium concentration to the transport sites of the ascending limb of the loop. The present studies were undertaken to determine if a relationship could be demonstrated between changes in the tubular reabsorption of sodium and changes in renal blood flow. It was observed that the increased excretion of sodium during isotonic expansion of the extracellular volume was uniformly associated with increased renal blood flow, independent of spontaneous changes in glomerular filtration rate. Furthermore, changes in the net tubular reabsorption of sodium, which were demonstrated both spontaneously during the course of saline loading and during controlled unilateral reductions of renal blood flow, were associated with inverse changes in that portion of renal plasma flow from which p-amino

Changes in Renal Blood Flow and Possibly the Intrarenal Distribution of Blood during the Natriuresis Accompanying Saline Loading in the Dog*