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It has been shown that cirrhosis of the liver is often accompanied by admixture of venous blood to the arterial blood, sometimes to a degree producing cyanosis (1-8). Two possible routes have been demonstrated morphologically: intrapulmonary arteriovenous shunts (2, 9), and channels connecting the portal circulation through esophageal and mediastinal veins with pulmonary veins (10, 11). The aim of the present study was to evaluate the physiological significance of these communications.

SOURCES OF VENOARTERIAL ADMIXTURE IN PORTAL HYPERTENSION*