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Many obese people consume most of their food within a relatively short period each day. These people eat little or no breakfast or lunch but eat voraciously during the evening (1, 2). Stunkard, Grace and Wolff (3) have used the term, "nighteating syndrome" to describe this pattern of eating and suggested that this pattern is the result of psychological factors. Some strains of rats can be trained to eat sufficient food in 1 hour each day to permit normal rates of growth (4) and studies of the respiratory quotients of these animals indicate increased lipogenesis (5). Tepperman and Tepperman (6) have shown that liver slices from rats trained in this way show increased incorporation of acetate1-C'4 and glucose-U-C14 in vitro, as well as a threeto fourfold increase in hexose monophosphate shunt dehydrogenase activity. However, lipogenesis in liver represents only a small fraction of the total lipid synthesis in the intact animal. Adipose tissue contributes the major portion (7). These observations coupled with the unusual eating pattern seen in many obese people prompted this study of adipose tissue and liver in rats allowed access to more food than they were able to consume for 2 hours out of each 24, for periods of 1 to 7 days. Studies were made of acetate-l-C'4 incorporation into lipids by adipose tissue and liver slices in vitro, free fatty acid (FFA) content of adipose tissue, glucose-6phosphate (G-6-P) dehydrogenase and 6-phosphogluconic (6-P-G) dehydrogenase activity of adipose tissue and liver homogenates and liver glycogen.

the journal of clinical investigation/the journal of clinical investigation

METABOLIC ADAPTATIONS TO A “STUFF AND STARVE” FEEDING PROGRAM. I. STUDIES OF ADIPOSE TISSUE AND LIVER GLYCOGEN IN RATS LIMITED TO A SHORT DAILY FEEDING PERIOD*