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The presence of baroreceptors in the walls of the cardiac chambers and the pulmonary vascular bed is now well established, and their function in the reflex control of the circulation has been the subject of a number of investigations (1-11). Action potentials have been recorded from afferent fibers originating in the heart and lungs (1, 2), and elevation of pressures within the ventricular chambers (3-9) and pulmonary vascular bed (4, 10, 11) has resulted in bradycardia (3-10), a decline in systemic arterial pressure (4-9, 11), and either apnea (5, 10) or hyperpnea (4, 11). In a previous report from this laboratory, evidence was presented that the carotid baroreceptors reflexly influence total venous return and systemic vascular volume (12). The present investigations were undertaken to determine whether or not these hemodynamic parameters are also modified by activation of intracardiac baroreceptors. Studies to localize these receptors within the heart were also carried out.

THE INFLUENCE OF INTRACARDIAC BARORECEPTORS ON VENOUS RETURN, SYSTEMIC VASCULAR VOLUME AND PERIPHERAL RESISTANCE