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It is now well established that the esterification of long chain fatty acids is an important step in their transport across the small intestinal mucosa before they appear in the lymph in chylomicrons. Evidence for this process has been well reviewed recently (1, 2). The main ester formed by the mucosa is triglyceride but a small proportion of the fatty acids is incorporated into phospholipids and cholesterol esters. We have recently reported some in vitro studies on the incorporation of long chain fatty acids into glycerides using homogenates of rat and human small intestine mucosa and have delineated some of the cofactor requirements involved (3). This work has now been extended to the intact mucosa of the rat. Observations are presented on factors influencing the uptake and esterification of palmitate-1-C4 and the incorporation of label into lipid from C14glucose. In addition, evidence is presented for the concept that conjugated bile salts directly stimulate glyceride metabolism in the intestinal

STUDIES ON LIPID METABOLISM IN THE SMALL INTESTINE WITH OBSERVATIONS ON THE ROLE OF BILE SALTS*†