English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Previous work has demonstrated that a severe burden is placed on the circulatory system of the dog by the surgical production of a long-sustained aortic-caval fistula (1). Circulatory distress is manifested by ascites, edema of all extremities and the lungs, together with a marked rise in left ventricular end-diastolic pressure, cardiac output, cardiac work, heart rate, and a depression of total peripheral resistance. The heart is at or near a state of cardiac decompensation, as indicated by the level of cardiac work and the response of the circulatory system to the infusion of whole blood or saline (1). Such infusions generally cause the cardiac output and cardiac work to decline and the left ventricular end-diastolic pressure to rise further, while the maximum level of cardiac work attained before infusion approximates that in the normal dog with massive infusion (1). To obtain further information on the physiological state of these dogs their response to rapid digitalization has been determined. This has been compared with the response of dogs without aortic-caval fistulas. The results of this study form the basis for this report.

The Hemodynamic Response to Lanatoside C of Dogs with Experimental Aortic-Caval Fistulas