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Rubidium, element number 37, is an alkalimetal found in small quantities in the earth's crust, and in trace amounts in human tissues (1). Listed immediately below potassium in the Group I elements of the periodic table, rubidium possesses many of potassium's physical, chemical, and physiological properties. These two elements have qualitatively similar effects on the contraction of the isolated heart (2), on the depolarization of nerve or muscle preparations (3, 4), and on the activity of certain intracellular enzyme systems (5, 6). The distribution of rubidium in human tissues resembles that of potassium (1, 7), and rubidium has been shown to be concentrated in certain plant cells (8) and in animal tissues (9, 10) with great rapidity. It has also been reported that addition of rubidium to a potassium-free diet will prevent the development in rats of most of the histological changes agsociated with potassium depletion ( 1) . The central purpose of the present work was to compare the effects of rubidium aidd potassium ions on acid-base balance in normal and in potassium-depleted rats. When given an alkaline-ash, potassium-deficient diet, rats develop an alkalosis (12), which can be rapidly corrected by administration of potassium chloride (13). The present data indicate that rubidium has an acidifying effect on extracellular fluid at least equal to, and under some circumstances clearly greater than, that of potassium.

THE ACIDIFYING EFFECT OF RUBIDIUM IN NORMAL AND POTASSIUM-DEFICIENT ALKALOTIC RATS 1