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tension. To determine the blood pressure effect of pharmacologic blockade of autonomic impulse transmission, TEAC in 3-5 cc. doses (300-500 mg.) was administered intravenously to all patients on from two to 12 occasions. All studies were conducted with the patients supine, and the injections were made rapidly, requiring no more than five seconds for administration. Observations of pupillary dilatation and abolition of sweating were accepted as clinical confirmation that autonomic blockade had been induced. In addition, in those patients who showed slight or moderate blood pressure depression following the drug, abolition of cold pressor reactivity was demonstrated to confirm autonomic block. Blood pressures were obtained by auscultation. The control blood pressure represents the mean of five or more measurements taken at oneminute intervals before TEAC administration and following stabilization of the pressure in the recumbent position. The lowest point to which the blood pressure descended in the first five minutes following injection has been termed the TEAC floor, and for the purposes of this study this has been accepted as indicating that portion of arteriolar tone which persists following the removal of active autonomic influence. Although multiple tests with TEAC were done on all patients, Table I includes only the results of the first test. The first experience with TEAC was selected since comparison was to be made with

EVALUATION OF NEUROGENIC CONTROL OF BLOOD PRESSURE IN HYPERTENSION WITH TETRAETHYLAMMONIUM CHLORIDE AND SPINAL ANESTHESIA 1