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The vascular and renal disorders of the last trimester of pregnancy are classified by the University Obstetrical Service in the following groups: specific toxemia, pre-existing essential hypertension, pre-existing glomerulonephritis and specific toxemia superimposed on pre-existing hypertension or nephritis (1). Specific toxemia is clinically characterized by the rather abrupt appearance of hypertension, usually in the last trimester of a previously normal pregnancy. This rise in blood pressure is typically accompanied by edema and proteinuria. Hematuria and azotemia are not seen, but the convulsions of eclampsia may occur. The postpartum course of the disease likewise helps to identify it. Edema subsides soon after delivery. Proteinuria usually disappears in the puerperium, although traces of protein in the urine may be present for weeks. Blood pressure falls to a normal level in the majority soon after delivery, but it is a striking feature of such an attack that in many instances it is followed by permanent hypertension. The present communication is an investigation of renal function in specific toxemia of pregnancy, based upon a study of inulin, phenol red and diodrast clearances and diodrast Tm. The inulin clearance is considered to be a measure of the rate of glomerular filtration, diodrast clearance a measure of effective renal blood flbw and diodrast Tm a measure of tubular excretory mass (2, 3), within the definitions and limitations accorded to these terms by Smith (4). Using these methods, Goldring et al. (5, 6, 7, 8) have demonstrated that renal ischemia of varying degree, associated with an increase in filtration fraction which is indicative of efferent arteriolar

THE FILTRATION RATE, EFFECTIVE RENAL BLOOD FLOW, TUBULAR EXCRETORY MASS AND PHENOL RED CLEARANCE IN SPECIFIC TOXEMIA OF PREGANANCY 1