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Nonarteritic anterior ischemic optic neuropathy (NAION) is the most common cause of sudden optic nerve-related vision loss in persons older than 50 in the United States. There currently is no treatment for this disorder. We previously showed that systemic administration of 15-deoxy, delta (12, 14) prostaglandin J2 (PGJ2) is neuroprotective in our rodent model of AION (rAION). In this study, we determined if a single intravitreal (IVT) injection of PGJ2 is neuroprotective after rAION, and if this method of administration is toxic to the retina, optic nerve, or both.

Sustained Neuroprotection From a Single Intravitreal Injection of PGJ2in a Rodent Model of Anterior Ischemic Optic Neuropathy

Sustained Neuroprotection From a Single Intravitreal Injection of PGJ₂in a Rodent Model of Anterior Ischemic Optic Neuropathy