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Hyperosmotic stress causes cell shrinkage, perturbs cell function, and damages DNA, resulting in cell cycle arrest and apoptosis. In the present study, the authors explore the mechanism involving hyperosmotic stress-induced activation of c-Jun/AP-1 through a novel Plk3 pathway in human corneal epithelial cells.

Hyperosmotic Stress-Induced Corneal Epithelial Cell Death through Activation of Polo-like Kinase 3 and c-Jun