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To investigate how OPA1 expression and distribution are altered by increased nitric oxide (NO) and whether aminoguanidine, a relative selective NO synthase (NOS)-2 inhibitor, can restore OPA1 expression and subsequently increase retinal ganglion cell (RGC) survival in ocular hypertensive rats.

Inducible Nitric Oxide Synthase-Mediated Alteration of Mitochondrial OPA1 Expression in Ocular Hypertensive Rats