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Cyclooxygenase (COX)-derived prostaglandin E(2) (PGE(2)) is a prevalent and established mediator of inflammation and pain in numerous tissues and diseases. Distribution and expression of the four PGE(2) receptors (EP1-EP4) can dictate whether PGE(2) exerts an anti-inflammatory or a proinflammatory and/or a proangiogenic effect. The role and mechanism of endogenous PGE(2) in the cornea, and the regulation of EP expression during a dynamic and complex inflammatory/reparative response remain to be clearly defined.

Selective Activation of the Prostaglandin E2Circuit in Chronic Injury-Induced Pathologic Angiogenesis

Selective Activation of the Prostaglandin E₂Circuit in Chronic Injury-Induced Pathologic Angiogenesis