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To define rod and cone function further in terms of visual cycle mechanism, the retinal phenotype resulting from Rpe65 (retinoid isomerase I) deficiency in Nrl(-)(/)(-) mice having a single class of photoreceptors resembling wild-type cones was characterized and outcomes of retinoid supplementation evaluated.

Nrl-Knockout Mice Deficient in Rpe65 Fail to Synthesize 11-cisRetinal and Cone Outer Segments