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The loss of atrial contractile function after cardioversion of atrial fibrillation (AF) contributes to the thromboembolic risk associated with AF. The newly developed blocker of the transient outward current (I(to)) and ultrarapid delayed rectifier current (I(Kur)) AVE0118 prolongs atrial action potential duration and might therefore enhance atrial contractility. We compared the ability of AVE0118 to restore atrial contraction after cardioversion of AF with the efficacy of conventional positive inotropic compounds in the goat model of AF.

circulation

AVE0118, Blocker of the Transient Outward Current ( I to ) and Ultrarapid Delayed Rectifier Current ( I Kur ), Fully Restores Atrial Contractility After Cardioversion of Atrial Fibrillation in the Goat