Open AccessEndothelial Nitric Oxide Synthase Deficiency Causes Collateral Vessel Rarefaction and Impairs Activation of a Cell Cycle Gene Network During Arteriogenesis
Author(s) -
Xuming Dai,
James E. Faber
Publication year - 2010
Publication title -
circulation research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 4.899
H-Index - 336
eISSN - 1524-4571
pISSN - 0009-7330
DOI - 10.1161/circresaha.109.212746
Subject(s) - arteriogenesis , nitric oxide synthase , nitric oxide , microbiology and biotechnology , endothelial stem cell , chemistry , medicine , cardiology , biology , angiogenesis , biochemistry , in vitro
The collateral circulation is tissue- and life-saving in obstructive arterial disease. Disappointing outcomes in clinical trials aimed at augmenting collateral growth highlight the need for greater understanding of collateral biology.
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