Interleukin-23 Deficiency Leads to Impaired Wound Healing and Adverse Prognosis After Myocardial Infarction | Zendy

Konstantinos Savvatis | Zendy; Kathleen Pappritz | Zendy; Peter Moritz Becher | Zendy; Diana Lindner | Zendy; Christin Zietsch | Zendy; HansDieter Volk | Zendy; Dirk Westermann | Zendy; HeinzPeter Schultheiss | Zendy; Carsten Tschöpe | Zendy

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Interleukin-23 Deficiency Leads to Impaired Wound Healing and Adverse Prognosis After Myocardial Infarction

Author(s) -

Konstantinos Savvatis,

Kathleen Pappritz,

Peter Moritz Becher,

Diana Lindner,

Christin Zietsch,

HansDieter Volk,

Dirk Westermann,

HeinzPeter Schultheiss,

Carsten Tschöpe

Publication year - 2013

Publication title -

circulation heart failure

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 3.352

H-Index - 104

eISSN - 1941-3297

pISSN - 1941-3289

DOI - 10.1161/circheartfailure.113.000604

Subject(s) - medicine , proinflammatory cytokine , inflammation , myocardial infarction , myofibroblast , wound healing , endocrinology , cytokine , immunology , fibrosis

CD4+ cells are implicated in the healing process after myocardial infarction (MI). We sought to investigate the role of interleukin-23 (IL-23) deficiency, a cytokine important in differentiation of CD4+ cells, in scar formation of the ischemic heart.

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