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Tumor necrosis factor superfamily ligands provoke a dilated cardiac phenotype signal through a common scaffolding protein termed tumor necrosis factor receptor-associated factor 2 (TRAF2); however, virtually nothing is known about TRAF2 signaling in the adult mammalian heart.

Tumor Necrosis Factor Receptor–Associated Factor 2 Signaling Provokes Adverse Cardiac Remodeling in the Adult Mammalian Heart