Genetic Variation in the SLC8A1 Calcium Signaling Pathway Is Associated With Susceptibility to Kawasaki Disease and Coronary Artery Abnormalities | Zendy

Chisato Shimizu | Zendy; Hariklia Eleftherohorinou | Zendy; Victoria Wright | Zendy; Jihoon Kim | Zendy; Martin P. Alphonse | Zendy; James C. Perry | Zendy; Rolando Cimaz | Zendy; David Burgner | Zendy; Nagib Dahdah | Zendy; Long Hoàng | Zendy; Chiea Chuen Khor | Zendy; Andrea Salgado | Zendy; Adriana H. Tremoulet | Zendy; Sonia Dávila | Zendy; Taco W. Kuijpers | Zendy; Martin L. Hibberd | Zendy; Todd A. Johnson | Zendy; Atsushi Takahashi | Zendy; Tatsuhiko Tsunoda | Zendy; Michiaki Kubo | Zendy; Toshihiro Tanaka | Zendy; Yoshihiro Onouchi | Zendy; Rae S. M. Yeung | Zendy; Lachlan Coin | Zendy; Michael Levin | Zendy; Jane C. Burns | Zendy

Open Access

Genetic Variation in the SLC8A1 Calcium Signaling Pathway Is Associated With Susceptibility to Kawasaki Disease and Coronary Artery Abnormalities

Author(s) -

Chisato Shimizu,

Hariklia Eleftherohorinou,

Victoria Wright,

Jihoon Kim,

Martin P. Alphonse,

James C. Perry,

Rolando Cimaz,

David Burgner,

Nagib Dahdah,

Long Hoàng,

Chiea Chuen Khor,

Andrea Salgado,

Adriana H. Tremoulet,

Sonia Dávila,

Taco W. Kuijpers,

Martin L. Hibberd,

Todd A. Johnson,

Atsushi Takahashi,

Tatsuhiko Tsunoda,

Michiaki Kubo,

Toshihiro Tanaka,

Yoshihiro Onouchi,

Rae S. M. Yeung,

Lachlan Coin,

Michael Levin,

Jane C. Burns

Publication year - 2016

Publication title -

circulation cardiovascular genetics

Language(s) - English

Resource type - Journals

eISSN - 1942-325X

pISSN - 1942-3268

DOI - 10.1161/circgenetics.116.001533

Subject(s) - kawasaki disease , genetic variation , coronary artery calcium , coronary artery disease , variation (astronomy) , medicine , disease , calcium , cardiology , artery , biology , genetics , gene , physics , astrophysics

Kawasaki disease (KD) is an acute pediatric vasculitis in which host genetics influence both susceptibility to KD and the formation of coronary artery aneurysms. Variants discovered by genome-wide association studies and linkage studies only partially explain the influence of genetics on KD susceptibility.

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