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In our article, we show less increased coronary heart disease (CHD) risk in familial hypercholesterolemia (FH) patients with a low-density lipoprotein receptor ( LDLR ) mutation and with the APOE4/E4 genotype.1 We give several possible explanations, including decreased trapping at the surface of hepatocytes when fewer LDL receptors are available. These scenarios could have been accompanied by higher serum ApoE levels, compared with the earlier described lower ApoE levels in APOE4/E4 genotyped individuals.2 However, in …

circulation. cardiovascular genetics

Response to the Letter by Singh et al Regarding “Apolipoprotein Isoform E4 Does Not Increase Coronary Heart Disease Risk in Carriers of Low-Density Lipoprotein Receptor Mutations”