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Inflammation contributes to hypertension-induced cardiac damage and fibrotic remodeling. Complement activation produces anaphylatoxins, which are major inflammatory effectors. Here, we investigated the role of complement anaphylatoxins in angiotensin II (Ang II)-induced cardiac remodeling.

Complement 5a Receptor Mediates Angiotensin II–Induced Cardiac Inflammation and Remodeling