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Serotonin, via 5-HT2 receptors, exerts an excitatory effect on CA1 neurons and may play a role in ischemia-induced excitotoxic damage. To evaluate the role of serotonin in ischemia, both neurochemical and histopathological studies were performed.

Ischemia-induced extracellular release of serotonin plays a role in CA1 neuronal cell death in rats.