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Carotid endarterectomy is currently guided by angiographic appearance on the assumption that the most stenotic lesion visible at angiography is likely to be the lesion from which future embolic events will arise. However, risk of plaque rupture, the most common cause of atherosclerosis-related thromboembolism, is dictated by the composition of the plaque, in particular the degree of inflammation. Angiography may, therefore, be an unreliable method of identifying vulnerable plaques. In this study, plaque inflammation was quantified before endarterectomy using the combination of 18F fluorodeoxyglucose positron (FDG)-emission tomography (PET) and high-resolution MRI (HRMRI).

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Identification of Culprit Lesions After Transient Ischemic Attack by Combined <sup>18</sup> F Fluorodeoxyglucose Positron-Emission Tomography and High-Resolution Magnetic Resonance Imaging

Identification of Culprit Lesions After Transient Ischemic Attack by Combined 18 F Fluorodeoxyglucose Positron-Emission Tomography and High-Resolution Magnetic Resonance Imaging

Identification of Culprit Lesions After Transient Ischemic Attack by Combined ¹⁸ F Fluorodeoxyglucose Positron-Emission Tomography and High-Resolution Magnetic Resonance Imaging