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The fetus is well known to be able to survive prolonged exposure to asphyxia with minimal injury compared with older animals. We and others have observed a rapid suppression of EEG intensity with the onset of asphyxia, suggesting active inhibition that may be a major neuroprotective adaptation to asphyxia. Adenosine is a key regulator of cerebral metabolism in the fetus.

Key Neuroprotective Role for Endogenous Adenosine A 1 Receptor Activation During Asphyxia in the Fetal Sheep

Key Neuroprotective Role for Endogenous Adenosine A ₁ Receptor Activation During Asphyxia in the Fetal Sheep