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Thrombin is activated in the cerebrospinal fluid (CSF) after a subarachnoid hemorrhage (SAH). However, the relationship between thrombin and cerebral vasospasm has not yet been fully established. The aim of this study was to investigate the possibility of thrombin as a causative factor for cerebral vasospasm and to delineate the signal transduction mechanism that results in thrombin-inducing sustained vasoconstriction in cerebral vasospasm.

Effects of Thrombin Inhibitor on Thrombin-Related Signal Transduction and Cerebral Vasospasm in the Rabbit Subarachnoid Hemorrhage Model