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Papaverine hydrochloride exhibited some ectopic impulse suppressor effect which was brief in duration and could not be maintained. Additional doses led to toxic reactions including increased ectopic activity. No consistent correlation was found between changes in blood pressure and decreases or increases in ectopic frequency. Vasodilator potency of drugs apparently bears no intimate relation to ectopic impulse suppressor effect.

Effects of Papaverine upon Ectopic Ventricular Tachycardia Produced by Myocardial Infarction