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Staphylococcus aureus sepsis is associated with significant myocardial dysfunction. Toll-like receptor 2 (TLR2) mediates the inflammatory response to S aureus and may trigger an innate immune response in the heart. We hypothesized that a TLR2 deficiency would attenuate S aureus-induced cardiac proinflammatory mediator production and the development of cardiac dysfunction.

Toll-Like Receptor 2 MediatesStaphylococcus aureus–Induced Myocardial Dysfunction and Cytokine Production in the Heart