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To the Editor:We read with interest the article by Ardilouze et al1 dealing with the possible roles of nitric oxide (NO) and adrenergic stimulation in the regulation of subcutaneous adipose tissue blood flow in humans. The results of their study demonstrated that NO seems to determine the absolute level of adipose tissue blood flow, whereas the postprandial enhancement of adipose tissue blood flow is highly dependent on β-adrenergic stimulation. The authors proposed that NO and β-adrenergic receptors may be major regulators of subcutaneous adipose tissue blood flow in humans.There is evidence that leptin, which is secreted from adipose tissue, might actively participate in the regulation of NO production. Nickola et al2 showed that leptin attenuated cardiac contraction in rat ventricular myocytes, possibly through an increased NO production. In a study we …

Role of Leptin in Regulating Nitric Oxide Production and Membrane Microviscosity