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Heart failure often complicates myocardial infarction (MI), and sarcoplasmic/endoplasmic reticulum Ca2+-ATPase (SERCA2a) is underexpressed in the failing myocardium. We examined the effect of preexisting cardiac SERCA2a protein overexpression on rat survival and left ventricular (LV) remodeling after MI.

circulation

Constitutive Cardiac Overexpression of Sarcoplasmic/Endoplasmic Reticulum Ca <sup>2+</sup> -ATPase Delays Myocardial Failure After Myocardial Infarction in Rats at a Cost of Increased Acute Arrhythmias

Constitutive Cardiac Overexpression of Sarcoplasmic/Endoplasmic Reticulum Ca 2+ -ATPase Delays Myocardial Failure After Myocardial Infarction in Rats at a Cost of Increased Acute Arrhythmias

Constitutive Cardiac Overexpression of Sarcoplasmic/Endoplasmic Reticulum Ca ²⁺ -ATPase Delays Myocardial Failure After Myocardial Infarction in Rats at a Cost of Increased Acute Arrhythmias