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Reactive oxygen species (ROS) may mediate pressure overload-induced myocardial hypertrophy. NADPH oxidase may be involved in this process, because its expression and activity are upregulated by pressure overload and because myocardial hypertrophy caused by a subpressor infusion of angiotensin is attenuated in mice deficient in the gp91phox catalytic subunit of NADPH oxidase.

Pressure Overload–Induced Myocardial Hypertrophy in Mice Does Not Require gp91 phox

Pressure Overload–Induced Myocardial Hypertrophy in Mice Does Not Require gp91 ^phox