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Identification of factors regulating myocardial structure and function is important to understand the pathogenesis of heart disease. We previously reported that 5-HT2B receptor ablation in mice leads to dilated cardiomyopathy. In this study, we investigated the pathological consequence of overexpressing 5-HT2B receptors in heart in vivo.

Overexpression of the Serotonin 5-HT 2B Receptor in Heart Leads to Abnormal Mitochondrial Function and Cardiac Hypertrophy

Overexpression of the Serotonin 5-HT _2B Receptor in Heart Leads to Abnormal Mitochondrial Function and Cardiac Hypertrophy