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The mitochondrial K(ATP) channel (mitoK(ATP)) has been implicated as an end effector or trigger of ischemic preconditioning (IP). Although a mitoK(ATP) opener, diazoxide, mimics IP, mechanisms for the cardioprotective action remain unclear.

Diazoxide Opens the Mitochondrial Permeability Transition Pore and Alters Ca 2+ Transients in Rat Ventricular Myocytes

Diazoxide Opens the Mitochondrial Permeability Transition Pore and Alters Ca ²⁺ Transients in Rat Ventricular Myocytes