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Oxidative stress (OS) induces smooth muscle cell apoptosis in the atherosclerotic plaque, leading to plaque instability and rupture. Heme oxygenase-1 (HO-1) exerts cytoprotective effects in the vessel wall. Recent evidence suggests that PKB/Akt may modulate HO-1 activity. This study examined the role of Akt in mediating the cytoprotective effects of HO-1 in OS-induced apoptosis of human aortic smooth muscle cells (HASMCs).

arteriosclerosis, thrombosis, and vascular biology

Protection of Human Vascular Smooth Muscle Cells From H <sub>2</sub> O <sub>2</sub> -Induced Apoptosis Through Functional Codependence Between HO-1 and AKT

Protection of Human Vascular Smooth Muscle Cells From H 2 O 2 -Induced Apoptosis Through Functional Codependence Between HO-1 and AKT

Protection of Human Vascular Smooth Muscle Cells From H ₂ O ₂ -Induced Apoptosis Through Functional Codependence Between HO-1 and AKT