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It is well established that reactive astrocytes express L-type calcium channels (LTCC), but their functional role is completely unknown. We have recently shown that reactive astrocytes highly express the CaV1.2 α1-subunit around β-amyloid (Aβ) plaques in an Alzheimer mouse model. The aim of the present study was to explore whether Aβ peptides may regulate the mRNA expression of all LTCC subunits in primary mouse astrocytes in culture.

pharmacology

Short- and Long-Term Treatment of Mouse Cortical Primary Astrocytes with β-Amyloid Differentially Regulates the mRNA Expression of L-Type Calcium Channels