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TGF-beta1 plays a major role in extracellular matrix (ECM) accumulation in tissue fibrosis. Connective tissue growth factor appears to play a critical role in this effect. Endoglin is a component of the transforming growth factor b (TGF-beta) receptor complex. Endoglin is upregulated by TGF-beta1, but its functional role in ECM regulation is unknown. Using rat myoblasts as a model system, we have assessed the role of endoglin on regulating CTGF expression and ECM synthesis and accumulation in the presence or absence of TGF-beta1.

cellular physiology and biochemistry

Endoglin Expression Regulates Basal and TGF-β1-induced Extracellular Matrix Synthesis in Cultured L&lt;sub&gt;6&lt;/sub&gt;E&lt;sub&gt;9&lt;/sub&gt; Myoblasts