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Antiestrogens are used to treat estrogen receptor (ER)-alpha-positive breast cancers and cause a p27-dependent G(1) arrest. Estrogen-bound ER recruits Src to mediate proteolysis of p27 and drive cell proliferation. Here, we tested the antitumor efficacy of combined Src and aromatase inhibition for ER-positive breast cancer.

Combined Src and Aromatase Inhibition Impairs Human Breast Cancer GrowthIn vivoand Bypass Pathways Are Activated in AZD0530-Resistant Tumors