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In an article in this issue of Clinical Cancer Research , Yasui et al. ([1][1]) suggest that cyclin D1 may play a role in maintaining vascular endothelial growth factor (VEGF) expression and that antisense cyclin D1 could be useful for targeting cancer cells and tumor vessels. Tumor growth and

Antisense to Cyclin D1 Inhibits VEGF-Stimulated Growth of Vascular Endothelial Cells: Implication of Tumor Vascularization