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In this study we demonstrated that Δ Caecm33  double mutant showed reduced biofilm formation and causes less damage to gingival mucosa tissues. This was confirmed by the reduced level of necrotic cells and Bax/Bcl2 gene expression as apoptotic markers. In contrast, parental and  Caecm33  mutant strains decreased basement membrane protein production (laminin 5 and type IV collagen). We thus propose that  ECM33  gene/protein represents a novel target for the prevention and treatment of infections caused by  Candida .

mediators of inflammation

Disruption of the<i>ECM33</i>Gene in<i>Candida albicans</i>Prevents Biofilm Formation, Engineered Human Oral Mucosa Tissue Damage and Gingival Cell Necrosis/Apoptosis

Disruption of theECM33Gene inCandida albicansPrevents Biofilm Formation, Engineered Human Oral Mucosa Tissue Damage and Gingival Cell Necrosis/Apoptosis