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myocardial infarction (MI) is a leading cause of death and disability globally that typically results from the abrupt occlusion of an epicardial coronary artery ([9][1]). As a result, cardiomyocytes distal to the occlusion are subject to oxygen and nutrient deprivation, which triggers mitochondrial

Mitigation of myocardial ischemia-reperfusion injury via HIF-1α-frataxin signaling