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To investigate the presence and functionality of oestrogen receptor alpha (ERalpha) in interleukin (IL)1beta-treated rabbit articular chondrocytes in culture, and to determine the mechanisms of 17beta oestradiol (E2) effects on IL1beta-induced inducible nitric oxide synthase (iNOS) expression.

Oestrogens inhibit interleukin 1 -mediated nitric oxide synthase expression in articular chondrocytes through nuclear factor- B impairment